What is Cerebral Infarction?
Cerebral infarction is also known as ischemic stroke, it is the destruction of the corresponding brain tissue after cerebral artery occlusion, which may be accompanied by bleeding. The pathogenesis is thrombosis or embolism, and the symptoms varies with the blood vessels involved. Cerebral infarction accounts for 70% – 80% of all stroke cases.
What Is the Etiology of Cerebral Infarction?
Cerebral infarction is caused by the sudden decrease or stop of blood flow in the local blood supply artery of brain tissue, resulting in cerebral tissue ischemia and hypoxia in the blood supply area, leading to brain tissue necrosis and softening, accompanied by clinical symptoms and signs of corresponding parts, such as hemiplegia, aphasia, and other neurological deficit symptoms.
Main factors
Hypertension, coronary heart disease, diabetes, overweight, hyperlipidemia, eating fat, and family history. It is more common in the middle-aged and old people aged 45-70.
What Are the Clinical symptoms of Cerebral Infarction?
The clinical symptoms of cerebral infarction are complex, it is related to the location of brain damage, the size of cerebral ischemic vessels, the severity of ischemia, whether there are other diseases before the onset, and whether there are diseases related to other important organs. In some mild cases, there may not be symptoms at all, that is, asymptomatic cerebral infarction Of course, there can also be recurrent limb paralysis or vertigo, that is, transient ischemic attack. In some severe cases, there will not only be limb paralysis, but even acute coma or death.
If the lesions affect the cerebral cortex, there can be epileptic seizures in the acute stage of cerebrovascular disease. Usually, the highest incidence is within 1 day after the disease, while cerebrovascular diseases with epilepsy as the first occurrence are rare.
How to Treat Cerebral Infarction?
Treatment of the disease should be aware of the treatment of hypertension, especially in the patients with lacunar infarction in their medical histories.
(1) Acute Period
a) Improve the blood circulation of cerebral ischemia area and promote the recovery of nerve function as soon as possible.
b) To relieve cerebral edema, patients with large and severe infarct areas can use dehydrating agents or diuretics.
c) Low molecular weight dextran can be used to improve microcirculation and reduce blood viscosity.
d) Diluted blood
f) Thrombolysis: streptokinase and urokinase.
g) Anticoagulation: use Heparin or Dicoumarin to prevent thrombus dilation and new thrombosis.
h) Dilation of blood vessels: It is generally believed that the effect of vasodilators is unstable. For severe patients with increased intracranial pressure, it can sometimes aggravate the condition, therefore, it’s nor recommended to use in the early stage.
(2) Recovery Period
Continue to strengthen the training of paralyzed limb function and speech function. Drugs should be used in conjunction with physical therapy and acupuncture.
Post time: Jan-05-2021